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51-40-1

中文名稱 L-去甲腎上腺素酒石酸氫鹽(酯)
英文名稱 L-NORADRENALINE BITARTRATE
CAS 51-40-1
EINECS 編號(hào) 200-095-0
分子式 C12H17NO9
MDL 編號(hào) MFCD00036384
分子量 319.26
MOL 文件 51-40-1.mol
更新日期 2024/12/23 09:09:21
51-40-1 結(jié)構(gòu)式 51-40-1 結(jié)構(gòu)式

基本信息

中文別名
(R)-4-(2-氨基-1-羥基乙基)-1,2-苯二酚重酒石酸鹽一水合物
重酒石酸去甲腎上腺素
L-去甲腎上腺素酒石酸氫鹽(酯)
L-去甲腎上腺素酒石酸氫鹽
DL-NOREPINEPHRINE (2,5,6, Α-D2,Β-D1) D6旽CL
英文別名
(-)-ARTERENOL BITARTRATE SALT
l-4-(2-amino-1-hydroxyethyl)-1,2-benzenediol bitartrate
L-4-(A-HYDROXY-B-AMINOETHYL)CATECHOL BITARTRATE
L-ARTERENOL BITARTRATE
L-NORADRENALINE BITARTRATE
L-(-)-NOREPINEPHRINE-(+)-BITARTRATE
L-NOREPINEPHRINE BITARTRATE
NORADRENALINE
NORADRENALINE BITARTRATE
NOREPINEPHRINE BITARTRATE
R(-)-4-(2-AMINO-1-HYDROXYETHYL)-1,2-BENZENEDIOL-(+)-TARTRATE
(-)-noradrenalineacidtartrate
(-)-noradrenalinebitartratemonohydrate
(-)-noradrenalinetartrate
(-)-norepinephrinetartrate
alpha-(aminomethyl)-3,4-dihydroxy-benzylalcoho(-)-benzylalcohotartrate(1:1)
Benzylalcohol,.alpha.-(aminomethyl)-3,4-dihydroxy-,(-)-,tartrate(1:1)
Levarterenolbitartrate
l-noradrenalinetartrate
noradrenaline,tartrate(1:1)
所屬類別
原料藥:抗休克血管活性藥

物理化學(xué)性質(zhì)

儲(chǔ)存條件Store at 4°C, protect from light
溶解度31.93mg/mL in DMSO
熔點(diǎn)102-104 °C
酸度系數(shù)(pKa)8.64(at 25℃)
水溶解性31.93mg/mL in Water
CAS 數(shù)據(jù)庫(kù)51-40-1(CAS DataBase Reference)

安全數(shù)據(jù)

危險(xiǎn)性符號(hào)(GHS)GHS hazard pictogramsGHS hazard pictograms
GHS08,GHS06
警示詞危險(xiǎn)
危險(xiǎn)性描述H360-H300-H330-H310
危險(xiǎn)品運(yùn)輸編號(hào)3249
危險(xiǎn)等級(jí)6.1(b)
包裝類別III
毒性LD50 ivn-rat: 210 mg/kg YACHDS 7,627,79

知名試劑公司產(chǎn)品信息

常見問(wèn)題列表

生物活性
Norepinephrine tartrate (Levarterenol tartrate) 是一種天然的應(yīng)激激素和神經(jīng)遞質(zhì),是 β1 選擇性的adrenergic receptor激動(dòng)劑,EC50值為 5.37 μM。
靶點(diǎn)

EC50: 5.37 μM (β 1 -selective adrenergic receptor).

體外研究

Norepinephrine (NE) bitartrate monohydrate is generally considered to be a β 1 -subtype selective adrenergic agonist. Norepinephrine (NE) also has direct activity at the β 2 -adrenoceptor in higher concentrations. Adipocytes from the inguinal fat pad (iWA) or the interscapular fat pad (BA) are isolated from neonatal wild-type C57BL/6J mice and cultured. To examine the effect of activating AT2 upon β-adrenergic signaling, cAMP production is first assessed in response to Norepinephrine (NE, 10 μM) with or without CGP (10 nM) co-treatment. Norepinephrine (NE) increases cAMP as expected in iWA, and CGP does not alter this effectNorepinephrine (NE) is also known to induce lipolysis, and liberated fatty acids are required to functionally activate UCP1 protein and to stimulate heat production. CREB phosphorylation at Ser133 is increased after Norepinephrine (NE) treatment and significantly attenuated with CGP co-treatment in mouse iWA.

RT-PCR

Cell Line: Subcutaneous preadipocytes Adipocytes.
Concentration: 10 μM.
Incubation Time: 6 hours.
Result: AT2 activation suppressed Norepinephrine induced UCP1 in white adipocytes (iWA)
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