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21593-77-1

中文名稱 S- 烯丙基別半胱氨酸,蒜氨酸
英文名稱 S-ALLYL-L-CYSTEINE
CAS 21593-77-1
分子式 C6H11NO2S
MDL 編號 MFCD00151975
分子量 161.22
MOL 文件 21593-77-1.mol
更新日期 2024/12/17 18:22:45
21593-77-1 結(jié)構(gòu)式 21593-77-1 結(jié)構(gòu)式

基本信息

中文別名
S- 烯丙基別半胱氨酸,蒜氨酸
S- 烯丙基-L-半胱氨酸
烯丙基-L-半胱氨酸
英文別名
CYSTEINE, S-ALLYL-L-
DEOXYALLIIN
(L)-3-(ALLYLSULFENYL)-ALANINE
L-DEOXYALLIIN
(R)-ALLYLTHIO-2-AMINOPROPIONIC ACID
S-ALLYL-L-CYSTEINE
s-allylcysteine
Allylcysteine
所屬類別
生物化工:半胱氨酸類衍生物

物理化學(xué)性質(zhì)

熔點(diǎn)235-236℃
沸點(diǎn)300℃
密度1.191
閃點(diǎn)135℃
儲存條件-20°C
溶解度H2O:>10mg/mL
酸度系數(shù)(pKa)2.07±0.10(Predicted)
形態(tài)粉末
顏色白色至米色
氣味 (Odor)at 0.10 % in propylene glycol. cooked roasted
香型roasted
旋光性 (optical activity)[α]/D -8 to -15°, c = 1 in H2O
JECFA Number1710
穩(wěn)定性感光
LogP1.31
CAS 數(shù)據(jù)庫21593-77-1(CAS DataBase Reference)

安全數(shù)據(jù)

危險性符號(GHS)GHS hazard pictograms
GHS07
警示詞警告
危險性描述H317
防范說明P280
危險品標(biāo)志Xi
危險類別碼43
安全說明36/37
WGK Germany3
RTECS號HA2466200
海關(guān)編碼2930.90.9250

知名試劑公司產(chǎn)品信息

常見問題列表

生物活性
S-Allyl-L-cysteine 是一種在大蒜中發(fā)現(xiàn)的有機(jī)硫化物,擁有多種生物活性,包括神經(jīng)營養(yǎng)活性,抗癌活性,抗炎活性等。
體外研究

It is found that S-Allyl-L-cysteine could protect against amyloid-protein (A)-and tunicamycin-induced cell death in differentiated PC12 cells. Simultaneously applied S-Allyl-L-cysteine (1 μM) suppresses the cell death induced by Aβ 25-35 and Aβ 1-40 in a concentration-dependent manner, and neuronal integrity is almost completely retained. Simultaneously applied S-Allyl-L-cysteine significantly decreases the Aβ-induced level of ROS. The TEAC value of S-Allyl-L-cysteine is lower than that of oxidized GSH, and no antioxidant activity is observed. Intracellular GSH levels remains unaffected by treatment of neurons with S-Allyl-L-cysteine for 24 h. Furthermore, the increase in caspase-12 protein expression is suppressed by simultaneously adding 1 μM S-Allyl-L-cysteine . S-Allyl-L-cysteine up to a concentration 1.0 mM does not exhibit any cytotoxic impact on morphology of myoblast and myotubes in culture observed under bright field microscope. TNF treatment leads to a significant decrease in the intracellular CK activity while S-Allyl-L-cysteine pre-treatment to TNF treated myotubes decreases the release of CK in media. S-Allyl-L-cysteine pre-treatment decreases the level of active form of this enzyme in S-Allyl-L-cysteine+TNF group. Similar observations are recorded at mRNA level for caspase-3. These results illustrate that S-Allyl-L-cysteine regulates apoptotic signals via suppressing the transcription and thus protein expression of caspase-3.

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