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GQ-16

GQ-16 ??? ???
?? ??:
870554-67-9
???:
GQ-16
???(??):
GQ-16;GQ-16 Exclusive;5Z-[(5-bromo-2-methoxyphenyl)methylene]-3-[(4-methylphenyl)methyl]-2,4-thiazolidinedione;2,4-Thiazolidinedione, 5-[(5-bromo-2-methoxyphenyl)methylene]-3-[(4-methylphenyl)methyl]-, (5Z)-;(5Z)-5-[(5-bromo-2-methoxyphenyl)methylidene]-3-[(4-methylphenyl)methyl]-1,3-thiazolidine-2,4-dione;PPAR,Inhibitor,reduced adipogenic actions,PPARγ,GQ-16,insulin sensitization,inhibit,Peroxisome proliferator-activated receptors,insulin Resistance,GQ16,GQ 16
CBNumber:
CB63152621
???:
C19H16BrNO3S
??? ??:
418.3
MOL ??:
870554-67-9.mol

GQ-16 ??

???
147 °C
?? ?
549.8±60.0 °C(Predicted)
??
1.522±0.06 g/cm3(Predicted)
?? ??
Store at -20°C
???
≤0.33mg/ml in DMSO;5mg/ml in dimethyl formamide
??? ??
??? ??
?? ?? (pKa)
-2.57±0.20(Predicted)
??
Off-white to light yellow

??

GQ-16 C??? ??, ??, ??

??

Full activation of peroxisome proliferator-activated receptor γ (PPARγ), e.g., using thiazolidinediones, effectively treats insulin resistance and type 2 diabetes but also commonly promotes weight gain, hyperphagia, and edema. GQ-16 is a partial agonist for PPARγ (Ki = 160 nM) which induces adipogenesis significantly less than the full activator rosiglitazone. Like rosiglitazone, GQ-16 reverses high fat diet-mediated impairments in insulin signaling. However, GQ-16 does not induce weight gain, hyperphagia, or edema. GQ-16 does not activate PPARα, PPARβ/δ, or RXRα.

GQ-16 ?? ?? ? ???

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GQ-16 ?? ??

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